Download Amyotrophic Lateral Sclerosis: A Guide for Patients and by Hiroshi Mitsumoto MD PDF

By Hiroshi Mitsumoto MD

This significant publication covers each point of dwelling with ALS, from scientific positive aspects of the ailment to an outline of symptom administration to knowing its analysis. significant sections care for overseeing therapy and rehabilitation, residing with ALS, the disease’s complex section, end-of-life matters, and assets. greatly revised and rewritten, this variation comprises new chapters at the most modern techniques to treating ALS signs, using noninvasive ventilators, multidisciplinary crew care, new directions being constructed by way of the yank Academy of Neurology for sufferers with ALS, using riluzole (Rilutek), and extra.

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Additional resources for Amyotrophic Lateral Sclerosis: A Guide for Patients and Families

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Cremer NE, Oshiro LS, Norris FH, et al. Cultures of tissues from patients with amyotrophic lateral sclerosis. Arch Neurol. 1973;29:331–333. 37. Roos RP, Viola MV, Wollmann R, et al. Amyotrophic lateral sclerosis with antecedent poliomyelitis. Arch Neurol. 1980;37:312–313. 38. Weiner LP, Stohlman SA, Davis RL. Attempts to demonstrate virus in amyotrophic lateral sclerosis. Neurology. 1980;30:1319–1322. 39. Wilson SAK. The amyotrophy of chronic lead poisoning: amyotrophic lateral sclerosis of toxic origin.

45. Ask-Upmark E. Amyotrophic lateral sclerosis observed in 5 persons after gastric resection. Gastroenterology. 1950;15:257–259. 46. Ask-Upmark E. Precipitating factors in the pathogenesis of amyotrophic lateral sclerosis. Acta Med Scand. 1961;170:717–723. 47. Siddique T, Figlewicz DA, Pericak-Vance MA, et al. Linkage of a gene causing familial amyotrophic lateral sclerosis to chromosome 21 and evidence of genetic-locus heterogeneity. N Engl J Med. 1991;324:1381–1384. 48. Rosen DR, Siddique T, Patterson D, et al.

24 Amyotrophic Lateral Sclerosis have no symptoms that they or their physician notice. It is not known how long this preclinical stage lasts before muscle weakness becomes clinically apparent. It depends on the rate of motor neuron degeneration and reinnervation. When detailed electrodiagnostic tests that measure the electrical activity of nerves and muscles are performed, neurologists may find evidence of extensive reinnervation, even when ALS is in a very early stage. The 2001 Aggarwal & Nicholson study from Australia mentioned above provides an important clue.

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